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Effect of dietary soy isoflavones on bone loss in ovariectomized rats SCIE Scopus
期刊论文 | 2018 , 17 (1) , 91-96 | TROPICAL JOURNAL OF PHARMACEUTICAL RESEARCH
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Purpose: To determine the effect of dietary soy isoflavone supplementation on bone loss in ovariectomized (OVX) rats. Methods: Forty-eight rats were assigned randomly to groups of OVX rats receiving soy isoflavones (20, 30, or 40 mg/kg of body weight daily), untreated OVX rats, or untreated intact rats. After 8 weeks, bone mineral density (BMD), mineral (Ca, P, Mn, Mg, and Zn) concentrations, and the expression of osteoblast-related genes were measured in femur tissue samples. Results: Eight weeks after OVX, there was a significant decrease in body weight, serum levels of osteocalcin, alkaline phosphatase, and oestradiol, BMD and mineral elements, as well as the expressions of Ctnnb1, Runx2, and Sp7 (all p < 0.05). These decreases were accompanied by reduced maximum load capacity of lumbar vertebrae. Daily supplementation with soy isoflavones dose-dependently ameliorated these effects (all p < 0.05). Western blotting revealed that these effects were likely due to the reversal of the OVX-induced decrease in Notch1 proteins in bone and muscle. Conclusion: Soy isoflavone treatment represents a potential therapy for preventing postmenopausal bone loss by stimulating the Notch signalling.

Keyword :

Isoflavones Mineral elements Alkaline phosphatase Bone loss Notch pathway

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GB/T 7714 Liang, Haidong , Yu, Fang , Yuan, Bo et al. Effect of dietary soy isoflavones on bone loss in ovariectomized rats [J]. | TROPICAL JOURNAL OF PHARMACEUTICAL RESEARCH , 2018 , 17 (1) : 91-96 .
MLA Liang, Haidong et al. "Effect of dietary soy isoflavones on bone loss in ovariectomized rats" . | TROPICAL JOURNAL OF PHARMACEUTICAL RESEARCH 17 . 1 (2018) : 91-96 .
APA Liang, Haidong , Yu, Fang , Yuan, Bo , Zhao, Zheng Nan , Tong, Chang Gui , Liu, Xue Hui et al. Effect of dietary soy isoflavones on bone loss in ovariectomized rats . | TROPICAL JOURNAL OF PHARMACEUTICAL RESEARCH , 2018 , 17 (1) , 91-96 .
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内侧半月板失稳诱发C57BL/6小鼠骨关节炎模型中关节腔微量注射方法的建立 Scopus CSCD PKU
期刊论文 | 2018 , (3) , 434-437,441 | 西安交通大学学报(医学版)
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目的 建立C57BL/6小鼠骨关节炎(OA)模型关节腔微量注射的图文操作流程.方法 24只雄性小鼠分假手术组及内侧半月板失稳术(DMM)组,显微操作下离断右膝内侧半月板胫副韧带(MMTL)诱发关节力学失稳,在OA模型基础上通过甲苯胺蓝关节腔注射验证微量给药的可行性,采用OARSI评分系统评估OA小鼠膝关节病理等级.结果 DMM组小鼠关节软骨番红O着色缺失,按关节分区评分,股骨内侧髁评分为3.417 0±0.642 1(n=12),内侧胫骨平台评分为9.583 0±0.811 5(n=12);假手术组对应部位评分依次为0.416 7±0.148 6(n=12),0.583 3±0.148 6(n=12).DMM组和假手术组OA病理评分相比较,股骨内侧髁(P=0.000 2)及胫骨平台(P<0.000 1)评分具有统计学差异.此外,区别于对照组关节腔注射生理盐水,微量注射甲苯胺蓝成功时可使透明软骨着色.结论 内侧半月板失稳术可诱导C57BL/6小鼠产生典型OA病损,OASRI评分提示内侧股骨髁及胫骨平台OA进展严重;可利用甲苯胺蓝验证小鼠OA模型中关节腔给药的重复性和可靠性.

Keyword :

OARSI评分 内侧半月板失稳术 骨关节炎 小鼠 关节腔微量注射

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GB/T 7714 冯萌 , 钱庄 , 李萌 et al. 内侧半月板失稳诱发C57BL/6小鼠骨关节炎模型中关节腔微量注射方法的建立 [J]. | 西安交通大学学报(医学版) , 2018 , (3) : 434-437,441 .
MLA 冯萌 et al. "内侧半月板失稳诱发C57BL/6小鼠骨关节炎模型中关节腔微量注射方法的建立" . | 西安交通大学学报(医学版) 3 (2018) : 434-437,441 .
APA 冯萌 , 钱庄 , 李萌 , 彭航 , 张婉 , 武淑芳 et al. 内侧半月板失稳诱发C57BL/6小鼠骨关节炎模型中关节腔微量注射方法的建立 . | 西安交通大学学报(医学版) , 2018 , (3) , 434-437,441 .
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Multiple congenital bilateral trigger digits in a 3-year-old child: a case report and literature review SCIE
期刊论文 | 2018 , 11 (10) , 11309-11312 | INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE
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Abstract :

Trigger finger is a common disorder characterized by clicking, catching, or loss of motion of the involved finger flexor tendon, and is associated with dysfunction and pain. Trigger digits in the pediatric population are relatively uncommon, while multiple congenital bilateral trigger fingers in children are extremely rare. In this paper, we present a 3.5-year-old boy with multiple congenital bilateral trigger digits. Triggering occurred in the proximal interphalangeal joints of the left middle and ring fingers and the right middle finger. The patient was not treated with any physical therapy before his presentation to our clinic. His symptoms completely resolved after surgical treatment with open division of the A1 pulleys. We believe that open release has satisfactory and encouraging results, and may be the preferred method of parents for trigger finger release.

Keyword :

congenital surgical management A1 pulley Trigger digit

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GB/T 7714 Feng, Dongxu , Wu, Shufang , Wang, Hao et al. Multiple congenital bilateral trigger digits in a 3-year-old child: a case report and literature review [J]. | INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE , 2018 , 11 (10) : 11309-11312 .
MLA Feng, Dongxu et al. "Multiple congenital bilateral trigger digits in a 3-year-old child: a case report and literature review" . | INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE 11 . 10 (2018) : 11309-11312 .
APA Feng, Dongxu , Wu, Shufang , Wang, Hao , Gao, Tianqi , Zhang, Jun , Zhu, Yangjun et al. Multiple congenital bilateral trigger digits in a 3-year-old child: a case report and literature review . | INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE , 2018 , 11 (10) , 11309-11312 .
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Plate fixation with autogenous bone grafting for longstanding humeral shaft nonunion A retrospective study of 6 cases SCIE PubMed
期刊论文 | 2018 , 97 (35) | MEDICINE
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Longstanding humeral shaft nonunion is uncommon because humeral shaft fractures often respond well to conservative and surgical treatments. However, when it occurs, the treatment of longstanding humeral shaft nonunion is challenging. This study is a retrospective analysis of the clinical and radiographic findings in a consecutive series of patients with longstanding humeral shaft nonunions who underwent locking compression plate (LCP) fixation and autogenous iliac crest bone grafting. Six patients were surgically treated at XI'an Hong Hui Hospital for longstanding humeral shaft nonunions between February 2011 and June 2015. Four patients were of synovial pseudarthrosis, 1 was atrophic, and 1 was hypertrophic. Follow-up was for at least 12 months after intervention. Standardized treatment included a thorough debridement, LCP and screw fixation, and autogenous iliac crest bone grafting. In 3 patients, a single plate was applied, and in the other 3 patients, double plates were used. The main outcome measurements were shoulder and elbow function (Constant and Murley scale, and Mayo elbow performance index [MEPI]) and the visual analog scale (VAS) for pain. In addition, all complications were documented. Our series included 6 male patients with an average age of 56.3 years and an average nonunion duration of 19.5 years. All patients had previously undergone at least 1 operation. At a mean of 26 months follow-up, all fractures had achieved solid union and none of the implants had evidence of loosening or breakage. Postoperative alignment was within 10 degrees of anatomic in 4 patients, 1 patient had 23 degrees of valgus angulation, and 1 patient had a posterior angulation of 12 degrees. Mean humeral shortening was 2.8 cm. The mean Constant and Murley joint function score was 88.3 points, the mean MEPI was 96.7 points, and the mean VAS was 0.7. All patients reported significant improvement in shoulder and elbow function, and each patient was able to resume work and was satisfied with the treatment. Plate fixation combined with autogenous iliac crest bone grafting is an excellent option for the treatment of longstanding humeral shaft nonunion.

Keyword :

autogenous bone grafting humerus locking compression plate nonunion longstanding

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GB/T 7714 Feng, Dongxu , Zhang, Jun , Zhu, Yangjun et al. Plate fixation with autogenous bone grafting for longstanding humeral shaft nonunion A retrospective study of 6 cases [J]. | MEDICINE , 2018 , 97 (35) .
MLA Feng, Dongxu et al. "Plate fixation with autogenous bone grafting for longstanding humeral shaft nonunion A retrospective study of 6 cases" . | MEDICINE 97 . 35 (2018) .
APA Feng, Dongxu , Zhang, Jun , Zhu, Yangjun , Wu, Shufang , Shan, Junping , Ye, Aiming et al. Plate fixation with autogenous bone grafting for longstanding humeral shaft nonunion A retrospective study of 6 cases . | MEDICINE , 2018 , 97 (35) .
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Plate fixation through an anterior approach for coronoid process fractures: A retrospective case series and a literature review. PubMed SCIE
期刊论文 | 2018 , 97 (36) , e12041 | Medicine
WoS CC Cited Count: 1
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Coronoid process fractures of the ulna are difficult to treat, and are associated with stiffness, recurrent instability, and pain. Hence, treatment of coronoid process fractures are challenging for surgeons. The purpose of this study was to report the clinical outcomes of an anterior surgical approach associated with plate fixation for Regan and Morrey type II or type III fractures of the coronoid process.We evaluated 16 consecutive patients who underwent surgical treatment for fracture of the coronoid process of the ulna from March 2012 to July 2016. Ten patients had a type II fracture, and 6 patients had a type III fracture. All patients underwent surgical treatment for coronoid process fracture through an anterior approach. While preserving the neurovascular structure, all fractures were treated with buttress plate fixation, maintaining the gap between brachial artery and median nerve. Each patient was treated with concentric reduction of both the ulnotrochlear and the radiocapitellar articulations, without any evidence of elbow instability, except 1 case, who showed some medial instability.At the final follow-up, solid osseous union was confirmed for all coronoid fractures. The average time to radiologic union was 16.3 weeks. The mean flexion-extension arc was 124.25 ± 12.12 degree, with a mean flexion contracture of 8.25 ± 4.36 degree, and further flexion of 132.5 ± 9.31 degree. The mean forearm rotation arc was 167.81 ± 10.49 degree. Fifteen patients achieved a functional arc of motion. The mean Mayo elbow performance score was 92.1 points, with 12 excellent cases and 4 good cases.Coronoid process fractures of the ulna can be treated successfully with plate fixation through an anterior surgical approach, which allows for accurate reduction and rigid internal fixation and early functional exercise, resulting in a reasonable outcome.

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GB/T 7714 Feng Dongxu , Zhang Xin , Jiang Yonghong et al. Plate fixation through an anterior approach for coronoid process fractures: A retrospective case series and a literature review. [J]. | Medicine , 2018 , 97 (36) : e12041 .
MLA Feng Dongxu et al. "Plate fixation through an anterior approach for coronoid process fractures: A retrospective case series and a literature review." . | Medicine 97 . 36 (2018) : e12041 .
APA Feng Dongxu , Zhang Xin , Jiang Yonghong , Zhu Yangjun , Wang Hao , Wu Shufang et al. Plate fixation through an anterior approach for coronoid process fractures: A retrospective case series and a literature review. . | Medicine , 2018 , 97 (36) , e12041 .
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Defective autophagy in osteoblasts induces endoplasmic reticulum stress and causes remarkable bone loss SCIE PubMed Scopus
期刊论文 | 2018 , 14 (10) , 1726-1741 | AUTOPHAGY
WoS CC Cited Count: 10 SCOPUS Cited Count: 9
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Macroautophagy/autophagy is a highly regulated process involved in the turnover of cytosolic components, however its pivotal role in maintenance of bone homeostasis remains elusive. In the present study, we investigated the direct role of ATG7 (autophagy related 7) during developmental and remodeling stages in vivo using osteoblast-specific Atg7 conditional knockout (cKO) mice. Atg7 cKO mice exhibited a reduced bone mass at both developmental and adult age. The trabecular bone volume of Atg7 cKO mice was significantly lower than that of controls at 5 months of age. This phenotype was attributed to decreased osteoblast formation and matrix mineralization, accompanied with an increased osteoclast number and the extent of the bone surface covered by osteoclasts as well as an elevated secretion of TNFSF11/RANKL (tumor necrosis factor [ligand] superfamily, member 11), and a decrease in TNFRSF11B/OPG (tumor necrosis factor receptor superfamily, member 11b [osteoprotegerin]). Remarkably, Atg7 deficiency in osteoblasts triggered endoplasmic reticulum (ER) stress, whereas attenuation of ER stress by administration of phenylbutyric acid in vivo abrogated Atg7 ablation-mediated effects on osteoblast differentiation, mineralization capacity and bone formation. Consistently, Atg7 deficiency impeded osteoblast mineralization and promoted apoptosis partially in DDIT3/CHOP (DNA-damage-inducible transcript 3)- and MAPK8/JNK1 (mitogen-activated protein kinase 8)-SMAD1/5/8-dependent manner in vitro, while reconstitution of Atg7 could improve ER stress and restore skeletal balance. In conclusion, our findings provide direct evidences that autophagy plays crucial roles in regulation of bone homeostasis and suggest an innovative therapeutic strategy against skeletal diseases.

Keyword :

osteoporosis bone mass bone formation ER stress Autophagy

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GB/T 7714 Li, Huixia , Li, Danhui , Ma, Zhengmin et al. Defective autophagy in osteoblasts induces endoplasmic reticulum stress and causes remarkable bone loss [J]. | AUTOPHAGY , 2018 , 14 (10) : 1726-1741 .
MLA Li, Huixia et al. "Defective autophagy in osteoblasts induces endoplasmic reticulum stress and causes remarkable bone loss" . | AUTOPHAGY 14 . 10 (2018) : 1726-1741 .
APA Li, Huixia , Li, Danhui , Ma, Zhengmin , Qian, Zhuang , Kang, Xiaomin , Jin, Xinxin et al. Defective autophagy in osteoblasts induces endoplasmic reticulum stress and causes remarkable bone loss . | AUTOPHAGY , 2018 , 14 (10) , 1726-1741 .
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Sirtuin-1 (SIRT1) stimulates growth-plate chondrogenesis by attenuating the PERK-eIF-2-CHOP pathway in the unfolded protein response EI SCIE PubMed Scopus
期刊论文 | 2018 , 293 (22) , 8614-8625 | JOURNAL OF BIOLOGICAL CHEMISTRY
WoS CC Cited Count: 7 SCOPUS Cited Count: 7
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The NAD(+)-dependent deacetylase sirtuin-1 (SIRT1) has emerged as an important regulator of chondrogenesis and cartilage homeostasis, processes that are important for physiological skeletal growth and that are dysregulated in osteoarthritis. However, the functional role and underlying mechanism by which SIRT1 regulates chondrogenesis remain unclear. Using cultured rat metatarsal bones and chondrocytes isolated from rat metatarsal rudiments, here we studied the effects of the SIRT1 inhibitor EX527 or of SIRT1 siRNA on chondrocyte proliferation, hypertrophy, and apoptosis. We show that EX527 or SIRT1 siRNA inhibits chondrocyte proliferation and hypertrophy and induces apoptosis. We also observed that SIRT1 inhibition mainly induces the PERK-eIF-2-CHOP axis of the endoplasmic reticulum (ER) stress response in growth-plate chondrocytes. Of note, EX527- or SIRT1 siRNA-mediated inhibition of metatarsal growth and growth-plate chondrogenesis were partly neutralized by phenylbutyric acid, a chemical chaperone that attenuates ER stress. Moreover, EX527-mediated impairment of chondrocyte function (i.e. of chondrocyte proliferation, hypertrophy, and apoptosis) was partly reversed in CHOP-/- cells. We also present evidence that SIRT1 physically interacts with and deacetylates PERK. Collectively, our findings indicate that SIRT1 deacetylates PERK and attenuates the PERK-eIF-2-CHOP axis of the unfolded protein response pathway and thereby promotes growth-plate chondrogenesis and longitudinal bone growth.

Keyword :

bone growth chondrocyte PERK unfolded protein response (UPR) chondrogenesis sirtuin 1 (SIRT1) growth plate endoplasmic reticulum stress (ER stress)

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GB/T 7714 Kang, Xiaomin , Yang, Wei , Wang, Ruiqi et al. Sirtuin-1 (SIRT1) stimulates growth-plate chondrogenesis by attenuating the PERK-eIF-2-CHOP pathway in the unfolded protein response [J]. | JOURNAL OF BIOLOGICAL CHEMISTRY , 2018 , 293 (22) : 8614-8625 .
MLA Kang, Xiaomin et al. "Sirtuin-1 (SIRT1) stimulates growth-plate chondrogenesis by attenuating the PERK-eIF-2-CHOP pathway in the unfolded protein response" . | JOURNAL OF BIOLOGICAL CHEMISTRY 293 . 22 (2018) : 8614-8625 .
APA Kang, Xiaomin , Yang, Wei , Wang, Ruiqi , Xie, Tianping , Li, Huixia , Feng, Dongxu et al. Sirtuin-1 (SIRT1) stimulates growth-plate chondrogenesis by attenuating the PERK-eIF-2-CHOP pathway in the unfolded protein response . | JOURNAL OF BIOLOGICAL CHEMISTRY , 2018 , 293 (22) , 8614-8625 .
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Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展
期刊论文 | 2017 , (12) , 2289-2292,2297 | 医学综述
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颗粒体上皮蛋白前体(PGRN)广泛分布于上皮细胞、免疫细胞、神经细胞等多种细胞中,具有多种功能.完整形式的PGRN可通过与肿瘤坏死因子α(TNF-α)结合,进而抑制TNF-α/TNF受体通路来发挥抗炎作用,而经蛋白酶加工后的PGRN通过增加促炎因子的产生而具有促炎效应.PGRN可作为抗炎因子参与早期胚胎发育和组织重塑,还可通过抑制炎症、促进自噬在肾脏疾病发生发展过程中发挥重要作用,此外,PGRN也可作为脂肪细胞因子参与肥胖和胰岛素抵抗等过程,参与肥胖和糖尿病的发生.

Keyword :

肥胖 糖尿病 颗粒体上皮蛋白前体 炎症 肾脏疾病

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GB/T 7714 廖梓安 , 余慧 , 孙宏志 et al. Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展 [J]. | 医学综述 , 2017 , (12) : 2289-2292,2297 .
MLA 廖梓安 et al. "Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展" . | 医学综述 12 (2017) : 2289-2292,2297 .
APA 廖梓安 , 余慧 , 孙宏志 , 武淑芳 . Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展 . | 医学综述 , 2017 , (12) , 2289-2292,2297 .
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Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展
期刊论文 | 2017 , (12) , 2289-2292+2297 | 医学综述
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肥胖 糖尿病 颗粒体上皮蛋白前体 炎症 肾脏疾病

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GB/T 7714 廖梓安 , 余慧 , 孙宏志 et al. Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展 [J]. | 医学综述 , 2017 , (12) : 2289-2292+2297 .
MLA 廖梓安 et al. "Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展" . | 医学综述 12 (2017) : 2289-2292+2297 .
APA 廖梓安 , 余慧 , 孙宏志 , 武淑芳 . Progranulin在糖脂代谢及肾脏疾病调控机制中的研究进展 . | 医学综述 , 2017 , (12) , 2289-2292+2297 .
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Melatonin protects chondrocytes from impairment induced by glucocorticoids via NAD(+)-dependent SIRT1 SCIE PubMed Scopus
期刊论文 | 2017 , 126 , 24-29 | STEROIDS | IF: 2.523
WoS CC Cited Count: 3 SCOPUS Cited Count: 3
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Intra-articular injection of glucocorticoids is used to relieve pain and inflammation in osteoarthritis patients, which is occasionally accompanied with the serious side effects of glucocorticoids in collagen-producing tissue. Melatonin is the major hormone released from the pineal gland and its beneficial effects: on cartilage has been suggested. In the present study, we investigated the protective role of melatonin on matrix degeneration in chondrocytes induced by dexamethasone (Dex). The chondrocytes isolated from mice knee joint were treated with Dex, melatonin, EX527 and siRNA targeted for SIRT6, respectively. Dex treatment induced the loss of the extracellular matrix, NAD(+)/NADH ratio and NADPH concentration in chondrocytes. Melatonin alone have no effect on the quantity of proteoglycans and collagen type Hal, however, the pretreatment of melatonin reversed the negative effects induced by Dex. Meanwhile, the significant decrease in NAD(+)/NADH ratio and NADPH concentration in Dex group were up-regulated by pretreatment of melatonin. Furthermore, it was revealed that inhibition of SIRT1 blocked the protective effects of melatonin. The enhancement of NAD(+)-dependent SIRT1 activity contributes to the chondroprotecfive effects of melatonin, which has a great benefit to prevent dexamethasone-induced chondrocytes impairment.

Keyword :

SIRT1 Osteoarthritis Glucocorticoids NAD(+) Melatonin Chondrocytes

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GB/T 7714 Yang, Wei , Kang, Xiaomin , Qin, Na et al. Melatonin protects chondrocytes from impairment induced by glucocorticoids via NAD(+)-dependent SIRT1 [J]. | STEROIDS , 2017 , 126 : 24-29 .
MLA Yang, Wei et al. "Melatonin protects chondrocytes from impairment induced by glucocorticoids via NAD(+)-dependent SIRT1" . | STEROIDS 126 (2017) : 24-29 .
APA Yang, Wei , Kang, Xiaomin , Qin, Na , Li, Feng , Jin, Xinxin , Ma, Zhengmin et al. Melatonin protects chondrocytes from impairment induced by glucocorticoids via NAD(+)-dependent SIRT1 . | STEROIDS , 2017 , 126 , 24-29 .
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